Among the features of tumours is usually that their cells possess lost the capability to differentiate; therefore these hormones possess useful properties to avoid cancer. Currently, retinoic acid and steroids are being utilized to take care of some types of leukaemia. A report led by the study group on Genes and Tumor of the Bellvitge Biomedical Study Institute shows that the increased loss of BRG1 gene implies too little response of cells to these hormones, and then the tumour may continue developing. Study results have already been released in the journal EMBO Molecular Medication. Related StoriesMU researchers effectively treat canines with DMD, plan for human scientific trialsApoE4-carrying males with Alzheimer's disease vulnerable to brain bleedsCHOP experts delay symptoms, expand lifespan in animal style of Batten diseaseBRG1 gene The IDIBELL analysis group on Genes and Cancer tumor led by Montse Sanchez-Cespedes discovered some years back that the BRG1 gene, a tumour suppressor, is inactivated in non-little cell lung cancers by genetic mutations.They intervene to destroy it. Since they are innocuous bacterias, although quite typical in the surroundings, and since they induce an immune reaction, they are the ideal bacteria scientists can use to study environmentally friendly factor contributing, together with the genetic factor, to cause multiple sclerosis. Related StoriesExperimental drug shows guarantee in mice with multiple sclerosisStudy displays how dietary essential fatty acids affect development and progression of multiple sclerosisRegular exercise benefits children with multiple sclerosis’Normally, T-cells cannot penetrate in to the Central Nervous System’, adds Rea, ‘because the hematoencephalic barrier prevents them from doing so. But the bacterium modifies the characteristics of the T-cells and enables them to overcome the barrier.